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Yoo T*, Cho H*, Lee J, Park H, Yoo YE, Yang E, Kim JY, Kim H, and Kim E. (2018).
GABA neuronal deletion of Shank3 exons 14–16 in mice suppresses striatal excitatory synaptic input and induces social and locomotor abnormalities.
Front Cell Neurosci 12: 341. (abstract)
Jung H*, Park H*, Choi Y, Kang H, Lee E, Kweon H, Roh JD, Ellegood J, Choi W, Kang J, Rhim I, Choi SY, Bae M, Kim SG, Lee J, Chung C, Yoo T, Park H, Kim Y, Ha S, Um SM, Mo J, Kwon Y, Mah W, Bae YC, Kim H, Lerch JP, Paik SB, and Kim E. (2018).
Sexually dimorphic behavior, neuronal activity, and gene expression in Chd8-mutant mice.
Nat Neurosci 21: 1218-1228. (abstract)
Lee S*, Lee E*, Kim R, Kim J, Lee S, Park H, Yang E, Kim H and Kim E. (2018).
Shank2 deletion in parvalbumin neurons leads to moderate hyperactivity, enhanced self-grooming, and suppressed seizure susceptibility in mice.
Front Mol Neurosci 11:209. (abstract)
Um SM, Ha S, Lee H, Kim J, Kim K, Shin W, Cho YS, Roh JD, Kang J, Yoo T, Noh Y, Choi Y, Bae YC, and Kim E. (2018).
NGL-2 Deletion Leads to Autistic-like Behaviors Responsive to NMDAR Modulation.
Cell Rep 23:3839-3851. (abstract)
Li Y*, Kim R*, Cho YS, Song WS, Kim D, Kim K, Roh JD, Chung C, Park H, Yang E, Kim SJ, Ko J, Kim H, Kim MH, Bae YC, and Kim E. (2018).
Lrfn2-mutant mice display suppressed synaptic plasticity and inhibitory synapse development and abnormal social communication and startle response.
J Neurosci 38:5872-5887. (abstract)
Lie E, Li Y, Kim R, and Kim E. (2018).
SALM/Lrfn family synaptic adhesion molecules.
Front Mol Neurosci 11:105. (abstract)
Kim R*, Kim J*, Chung C*, Ha S, Lee S, Lee E, Yoo YE, Kim W, Shin W, Kim E. (2018).
Cell type-specific Shank2 deletion in mice leads to differential synaptic and behavioral phenotypes.
J Neurosci 38:4076-4092.
Ko J and Kim E. (2018).
Special issue on synapse assembly, neural circuit development, and brain disorders. 
Exp Mol Med 50:17. (abstract)
Bae YS, Yoon SH, Han JY, Woo J, Cho YS, Kwon SK, Bae YC, Kim D, Kim E, Kim MH. (2018).
Deficiency of aminopeptidase P1 causes behavioral hyperactivity, cognitive deficits, and hippocampal neurodegeneration.
Genes Brain Behav  17:126-138. (abstract)