Ph.D. Alumni마 원 / Won Mah / Ph.D. / 2006-03 ~ 2013-08 

11. Chung C*, Ha S*, Kang H, Lee J, Um SM, Yan H, Yoo YE, Yoo T, Jung H, Lee D, Lee E, Lee S, Kim J, Kim R, Kwon Y, Kim W, Kim H, Duffney L, Kim D, Mah W, Won H, Mo S, Kim JY, Lim CS, Kaang BK, Boeckers TM, Chung Y, Kim H, Jiang YH, and Kim E. (2019).
Early correction of NMDAR function improves autistic-like behaviors in adult Shank2–/– mice.
Biol Psychiatry 85:534-543. (abstract)

10. Jung H*, Park H*, Choi Y, Kang H, Lee E, Kweon H, Roh JD, Ellegood J, Choi W, Kang J, Rhim I, Choi SY, Bae M, Kim SG, Lee J, Chung C, Yoo T, Park H, Kim Y, Ha S, Um SM, Mo J, Kwon Y, Mah W, Bae YC, Kim H, Lerch JP, Paik SB, and Kim E. (2018).
Sexually dimorphic behavior, neuronal activity, and gene expression in Chd8-mutant mice.
Nat Neurosci 21: 1218-1228. (abstract)

9. Lie E, Ko JS, Choi SY, Roh JD, Cho YS, Noh R, Kim D, Li Y, Kang H, Cho TY, Nam J, Mah W, Lee D, Lee SG, Kim HM, Kim H, Choi SY, Um JW, Kang MG, Bae YC, Ko J, and Kim E. (2016).
SALM4 suppresses excitatory synapse development by cis-inhibiting trans-synaptic SALM3–LAR adhesion.
Nat Commun 7:12328. (abstract)

8. Choi Y*, Nam J*, Whitcomb DJ*, Song YS*, Kim D, Jeon S, Um JW, Lee SG, Woo J, Kwon S, Li Y, Mah W, Kim HM, Ko J, Cho K, and Kim E. (2016).
SALM5 trans-synaptically interacts with LAR-RPTPs in a splicing-dependent manner to regulate synapse development.
Sci Rep 6:26676. (abstract)

7. Jang S*, Oh D*, Lee Y*, Hosy E*, Shin H, van Riesen C, Whitcomb D, Warburton JM, Jo J, Kim D, Kim SG, Um SM, Kwon SK, Kim MH, Roh JD, Woo J, Jun H, Lee D, Mah W, Kim H, Kaang BK, Cho K, Rhee JS, Choquet D, and Kim E. (2016).
Synaptic adhesion molecule IgSF11 regulates synaptic transmission and plasticity.
Nat Neurosci 19:84-93. (abstract)

6. Li Y*, Zhang P*, Choi TY, Park SK, Park H, Lee EJ, Lee D, Roh JD, Mah W, Kim R, Kim Y, Kwon H, Bae YC, Choi SY, Craig AM, and Kim E. (2015).
Splicing-dependent Trans-synaptic SALM3-LAR-RPTP Interactions Regulate Excitatory Synapse Development and Locomotion.
Cell Rep 12:1618-1630. (abstract)

5. Won H, Mah W, and Kim, E. (2013).
Autism spectrum disorder causes, mechanisms, and treatments: focus on neuronal synapses.
Front Mol Neurosci 6:19. (abstract)

4. Won H*, Lee HR*, Gee HY*, Mah W*, Kim JI*, Lee J, Ha S, Chung C, Jung ES, Cho YS, Park SG, Lee JS, Lee K, Kim D, Bae YC, Kaang BK, Lee MG, and Kim E. (2012).
Autistic-like social behavior in Shank2-mutant mice improved by restoring NMDA receptor function.
Nature 486:261-265. (abstract) (Nature Reviews Drug Discovery) (SFARI) (Nature Reviews Neuroscience)

3. Nam J, Mah W, and Kim E. (2011).
The SALM/Lrfn family of leucine-rich repeat-containing cell adhesion molecules.
Semin Cell Dev Biol 22:492-498. (abstract)

2. Won H*, Mah W*, Kim E, Kim JW, Hahm EK, Kim MH, Cho S, Kim J, Jang H, Cho SC, Kim BN, Shin MS, Seo J, Jeong J, Choi SY, Kim D, Kang C, and Kim E. (* co-first author). (2011).
GIT1 is associated with attention deficit/hyperactivity disorder (ADHD) and ADHD-like behaviors in mice.
Nat Med 17:566-572. (abstract) (Nature Asia-Pacific) (News and Views) (Podcast) (Spoonful of Medicine) (Nature SciBx) 

1. Mah W*, Ko J*, Nam J*, Han K, Chung W and Kim E. (* co-first author). (2010). Selected SALM family proteins regulate synapse formation.
J Neurosci 30:5559-5568. (abstract) (pdf) (This Week in The Journal)