| Year | Number | Authors / Title / Journal |
| 2015 | 125 | Lee D, Yamamoto Y, Kim E, and Tanaka-Yamamoto K. (2015). |
| 2015 | 124 | Park H, Yang J, Kim R, Li Y, Lee Y, Lee C, Park J, Lee D, Kim H, and Kim E. (2015). Mice lacking the PSD-95-interacting E3 ligase, Dorfin/Rnf19a, display reduced adult neurogenesis, enhanced long-term potentiation, and impaired contextual fear conditioning. Sci Rep 5:16410. (abstract) |
| 2015 | 123 | Li Y*, Zhang P*, Choi TY, Park SK, Park H, Lee EJ, Lee D, Roh JD, Mah W, Kim R, Kim Y, Kwon H, Bae YC, Choi SY, Craig AM, and Kim E. (2015). Splicing-dependent Trans-synaptic SALM3-LAR-RPTP Interactions Regulate Excitatory Synapse Development and Locomotion. Cell Rep 12:1618. (abstract) |
| 2015 | 122 | Choi SY, Han K, Cutforth T, Chung W, Park H, Lee D, Kim R, Kim MH, Choi Y, Shen K, and Kim E. (2015). |
| 2015 | 121 | Whitcomb D*, Hogg E*, Regan P, Piers T, Narayan P, Whitehead G, Winters B, Kim DH, Kim E, St George-Hyslop P, Klenerman D, Collingridge G, Jo J, and Cho K. (2015). Intracellular oligomeric amyloid-beta rapidly regulates GluA1 subunit of AMPA receptor in the hippocampus. Sci Rep 5:10934. (abstract) |
| 2015 | 120 | Kang J and Kim E. (2015). Suppression of NMDA receptor function in mice prenatally exposed to valproic acid improves social deficits and repetitive behaviors. Front Mol Neurosci 8:17. (abstract) |
| 2015 | 119 | Lee EJ, Lee H, Huang TNE, Chung C, Shin W, Kim K, Koh JY, Hsueh YP, and Kim E. (2015). Trans-synaptic zinc mobilization improves social interaction in two mouse models of autism through NMDAR activation. Nat Commun 6:7168. (abstract) |
| 2015 | 118 | Varea O, Martin-de-Saavedra MD, Kopeikina KJ, Schürmann B, Fleming H, Patel J, Bach A, Jang S, Peles E, Kim E, and Penzes P. (2015). Synaptic abnormalities and cytoplasmic glutamate receptor aggregates inCntnap2/Caspr2 knockout neurons. Proc Natl Acad Sci USA 112:6176. (abstract) |
| 2015 | 117 | Lee J, Chung C, Ha S, Lee D, Kim DY, Kim H, and Kim E. (2015). Shank3-mutant mice lacking exon 9 show altered excitation/inhibition balance, enhanced rearing, and spatial memory deficit. Front Cell Neurosci 9:94. (abstract) |
| 2015 | 116 | Chung W*, Choi SY*, Lee E*, Park H*, Kang J, Park H, Choi Y, Lee D, Park SG, Kim R, Cho YS, Choi J, Kim MH, Lee JW, Lee S, Rhim I, Jung MH, Kim D, Bae YC, and Kim E. (2015). Social deficits in IRSp53 mutant mice improved by NMDAR and mGluR5 suppression. Nat Neurosci 18:435. (abstract) |
| 2015 | 115 | Ylikallio E, Kim D, Isohanni P, Auranen M, Lönnqvist T, Kim E, and Tyynismaa H. (2015). Dominant transmission of de novo KIF1A motor domain variant underlying pure spastic paraplegia. Eur J Hum Genet 23:1427. (abstract) |
| 2015 | 114 | Lee EJ, Choi SY, and Kim E. (2015). |
| 2015 | 113 | Lee JR, Srour M, Kim D, Hamdan FF, Lim SH, Brunel-Guitton C, Décarie JC, Rossignol E, Mitchell GA, Schreiber A, Moran R, Van Haren K, Richardson R, Nicolai J, Oberndorff KM, Wagner JD, Boycott KM, Rahikkala E, Junna N, Tyynismaa H, Cuppen I, Verbeek NE, Stumpel CT, Willemsen MA, de Munnik SA, Rouleau GA, Kim E, Kamsteeg EJ, Kleefstra T, and Michaud JL. (2015). De novo mutations in the motor domain of KIF1A cause cognitive impairment, spastic paraparesis, axonal neuropathy and cerebellar atrophy. Hum Mutat 36:69. (abstract) |
